Search results for " β-thalassemia"

showing 3 items of 3 documents

Hematopoietic stem cell function in b-thalassemia is impaired and is rescued by targeting the bone marrow niche

2020

Abstract Hematopoietic stem cells (HSCs) are regulated by signals from the bone marrow (BM) niche that tune hematopoiesis at steady state and in hematologic disorders. To understand HSC-niche interactions in altered nonmalignant homeostasis, we selected β-thalassemia, a hemoglobin disorder, as a paradigm. In this severe congenital anemia, alterations secondary to the primary hemoglobin defect have a potential impact on HSC-niche cross talk. We report that HSCs in thalassemic mice (th3) have an impaired function, caused by the interaction with an altered BM niche. The HSC self-renewal defect is rescued after cell transplantation into a normal microenvironment, thus proving the active role of…

MaleStromal cellImmunologybone marrow mice thalassemia hematopoietic stem cells transplantation parathyroid hormoneSettore MED/08 - Anatomia PatologicaBiochemistryBone remodelingMiceBone MarrowmedicineAnimalsHumansOsteopontinStem Cell NicheHematopoietic stem cell β-thalassemia the bone marrow nichebiologybeta-ThalassemiaHematopoietic stem cellCell BiologyHematologyHematopoietic Stem CellsHematopoiesisMice Inbred C57BLTransplantationHaematopoiesismedicine.anatomical_structurebiology.proteinCancer researchFemaleBone marrowStem cell
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Glutathione S Transferase Polymorphisms Influence on Iron Overload in β-Thalassemia Patients

2013

In patients with β-thalassemia iron overload that leads to damage to vital organs is observed. Glutathione S transferase (GST) enzymes have an antioxidant role in detoxification processes of toxic substances. This role is determined genetically. In this study, we correlated GSTT1 and GSTM1 genotypes with iron overload measured with direct and indirect non-invasive methods; in particular, we used serum ferritin and signal intensity of the magnetic resonance image (MRI) in 42 patients with β-thalassemia, which were regularly subjected to chelation and transfusion therapy. Multiplex polymerase chain reaction was used to determine the genotype. The loss of both alleles leads to a decreased valu…

medicine.medical_specialtyThalassemiaglutathione S transferases; β-thalassemia; iron overloadBiologymedicine.diseaseglutathione S transferases β-thalassemia iron overload.EndocrinologyGlutathione S-transferaseBiochemistryInternal medicinemedicinebiology.proteinDiseases of the blood and blood-forming organsRC633-647.5Thalassemia Reports
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Efficacy of Rapamycin as Inducer of Hb F in Primary Erythroid Cultures from Sickle Cell Disease and β-Thalassemia Patients.

2015

Phenotypic improvement of hemoglobinopathies such as sickle cell disease and β-thalassemia (β-thal) has been shown in patients with high levels of Hb F. Among the drugs proposed to increase Hb F production, hydroxyurea (HU) is currently the only one proven to improve the clinical course of these diseases. However, Hb F increase and patient's response are highly variable, indicating that new pharmacological agents could be useful for patients not responding to HU or showing a reduction of response during long-term therapy. In this study we evaluated the efficacy of rapamycin, a lypophilic macrolide used for the prevention of acute rejection in renal transplant recipients, as an inducer of Hb…

rapamycin β-Thalassemia
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